Tension pneumothorax in a ventilated patient

Tension pneumothorax in a ventilated patient

By Dr. Shannon KendrickUpdated September 2026

The case

A 62-year-old man with severe community-acquired pneumonia was intubated and started on positive-pressure ventilation in intensive care 30 minutes ago. He has ischaemic heart disease and is on a low-dose noradrenaline (norepinephrine) infusion. Shortly after the ventilator settings were increased for worsening oxygenation, he became acutely more hypoxic and hypotensive: heart rate 130/min, blood pressure 72/40 mmHg (previously 95/60), oxygen saturation 82% on 100% oxygen. The right hemithorax is hyper-resonant with absent breath sounds, the neck veins are distended and the trachea is deviated to the left. Arterial blood gas on 100% oxygen: pH 7.22, PaCO2 8.5 kPa, PaO2 6.0 kPa. A portable chest X-ray would take several minutes. What is the most appropriate immediate action?

Options

  1. ANeedle decompression, then intercostal catheter
  2. BIncrease noradrenaline and give fluids
  3. CPortable chest X-ray to confirm
  4. DCT pulmonary angiogram to exclude embolism
  5. EBag-valve ventilation with oxygen

Think it through before you read on. Which single option is best, and why are the other four wrong?

Show the answer and explanation

Answer

A. Needle decompression, then intercostal catheter

Why this is the right answer

This is a tension pneumothorax. Air enters the pleural space with each breath and cannot leave. Pressure builds, the lung collapses, the mediastinum is pushed away, and venous return to the heart is blocked. The result is obstructive shock: hypotension, tachycardia, distended neck veins and profound hypoxia. Positive-pressure ventilation, especially after an increase in pressures, is a classic cause.

The diagnosis is clinical. Tracheal deviation is a late sign; absent breath sounds and hyper-resonance on one side with sudden cardiovascular collapse are enough. No imaging should be obtained before treatment, because the patient may arrest during the delay.

Immediate decompression is required. In an adult, a large-bore cannula is inserted in the fourth or fifth intercostal space in the anterior axillary line (the site now preferred over the second intercostal space mid-clavicular line, because chest wall thickness there often exceeds cannula length). A rush of air confirms the diagnosis. Alternatively, in a ventilated patient, a finger thoracostomy can be done directly. Decompression is a temporary measure and must be followed at once by a formal intercostal catheter (chest drain).

Therapeutic Guidelines and trauma teaching (EMST/ATLS) place this in the 'breathing' step of the primary survey, before circulation is addressed.

Vasopressors and fluid cannot overcome a mechanical obstruction to venous return. Continuing high-pressure ventilation without decompression drives more air into the pleural space and can cause cardiac arrest.

Why the other options are wrong

B

Fluids and pressors cannot relieve obstructive shock; the pleural pressure must be released.

C

X-ray confirmation delays life-saving treatment in an obvious clinical diagnosis.

D

CT is impossible in a crashing patient and the findings already explain the collapse.

E

More positive-pressure ventilation without decompression pushes more air into the pleural space and may cause arrest.

High-yield takeaway

Tension pneumothorax is a clinical diagnosis: decompress immediately (fifth intercostal space, anterior axillary line) and then insert a chest drain, never wait for an X-ray.

Reference: Therapeutic Guidelines: Emergency / EMST (Early Management of Severe Trauma) course principles (2023)

Common questions

Where should needle decompression be done?

The fourth or fifth intercostal space in the anterior axillary line, which is now preferred over the second intercostal space.

Is a chest X-ray ever appropriate first?

Not in a patient who is collapsing. The diagnosis is clinical and delay can cause cardiac arrest.

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